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Center Publications

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Ibrahim, S., et al. “Β-Cell Pre-Mir-21 Induces Dysfunction And Loss Of Cellular Identity By Targeting Transforming Growth Factor Beta 2 (Tgfb2) And Smad Family Member 2 (Smad2) Mrnas.”. Molecular Metabolism, p. 101289.
Indiana
Hu, W., et al. “Individual-Specific Functional Epigenomics Reveals Genetic Determinants Of Adverse Metabolic Effects Of Glucocorticoids.”. Cell Metabolism, pp. 1592-1609.e7.
Penn
Reynolds, E. L., et al. “The Determinants Of Complication Trajectories In American Indians With Type 2 Diabetes.”. Jci Insight.
Michigan
Wibowo, M. C., et al. “Reconstruction Of Ancient Microbial Genomes From The Human Gut.”. Nature, pp. 234-239.
Joslin
Friedlander, M. S. H., et al. “Pancreatic Pseudoislets: An Organoid Archetype For Metabolism Research.”. Diabetes, pp. 1051-1060.
Stanford
Angueira, A. R., et al. “Defining The Lineage Of Thermogenic Perivascular Adipose Tissue.”. Nature Metabolism, pp. 469-484.
Penn
Joslin
Callahan, M. L., et al. “Association Of Weight Status And Carbohydrate Intake With Gestational Weight Gain.”. Clinical Obesity, p. e12455.
UAB
Muller, Y. D., et al. “The Cd28-Transmembrane Domain Mediates Chimeric Antigen Receptor Heterodimerization With Cd28.”. Frontiers In Immunology, p. 639818.
UCSF
Sutton, A. K., et al. “Melanocortin 3 Receptor-Expressing Neurons In The Ventromedial Hypothalamus Promote Glucose Disposal.”. Proceedings Of The National Academy Of Sciences Of The United States Of America.
Michigan
Saunders, D. C., et al. “Coordinated Interactions Between Endothelial Cells And Macrophages In The Islet Microenvironment Promote Β Cell Regeneration.”. Npj Regenerative Medicine, p. 22.
Vanderbilt
Smith, C. D., et al. “Genetically Increasing Flux Through Β-Oxidation In Skeletal Muscle Increases Mitochondrial Reductive Stress And Glucose Intolerance.”. American Journal Of Physiology. Endocrinology And Metabolism.
Einstein