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Center Publications

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Kim, M. -H., et al. “Leprb+ Cell-Specific Deletion Of Slug Mitigates Obesity And Nonalcoholic Fatty Liver Disease In Mice.”. The Journal Of Clinical Investigation.
Michigan
Prasad, R., et al. “Maintenance Of Enteral Ace2 Prevents Diabetic Retinopathy In Type 1 Diabetes.”. Circulation Research, pp. e1-e21.
UAB
Mazor, R., et al. “Mesenteric Fat Cryolipolysis Attenuates Insulin Resistance In The Ossabaw Swine Model Of The Metabolic Syndrome.”. Surgery For Obesity And Related Diseases : Official Journal Of The American Society For Bariatric Surgery.
Indiana
Balikcioglu, P. G., et al. “Branched-Chain Α-Keto Acids And Glutamate/Glutamine: Biomarkers Of Insulin Resistance In Childhood Obesity.”. Endocrinology, Diabetes & Metabolism.
North Carolina
Hart, P. A., et al. “A Reduced Pancreatic Polypeptide Response Is Associated With New Onset Pancreatogenic Diabetes Versus Type 2 Diabetes.”. The Journal Of Clinical Endocrinology And Metabolism.
Indiana
Chang, C. A., et al. “Curative Islet And Hematopoietic Cell Transplantation In Diabetic Mice Without Toxic Bone Marrow Conditioning.”. Cell Reports, p. 111615.
Stanford
Shrestha, N., et al. “Integration Of Er Protein Quality Control Mechanisms Defines Β-Cell Function And Er Architecture.”. The Journal Of Clinical Investigation.
Michigan
Moore, T. M., et al. “Parkin Regulates Adiposity By Coordinating Mitophagy With Mitochondrial Biogenesis In White Adipocytes.”. Nature Communications, p. 6661.
UCSD-UCLA
Dickerson, M. T., et al. “G Protein-Coupled Receptor Inhibition Of Beta-Cell Electrical Excitability And Insulin Secretion Depends On Na/K Atpase Activation.”. Nature Communications, p. 6461.
Vanderbilt
Du, W., et al. “Pharmacological Conversion Of Gut Epithelial Cells Into Insulin-Producing Cells Lowers Glycemia In Diabetic Animals.”. The Journal Of Clinical Investigation.
Columbia
Hepler, C., et al. “Time-Restricted Feeding Mitigates Obesity Through Adipocyte Thermogenesis.”. Science (New York, N.y.), pp. 276-284.
Chicago
Park, J., et al. “Activation Of The Insulin Receptor By An Insulin Mimetic Peptide.”. Nature Communications, p. 5594.
Columbia