Center Publications

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Ramirez, A. K., et al. “Integrating Extracellular Flux Measurements And Genome-Scale Modeling Reveals Differences Between Brown And White Adipocytes.”. Cell Reports, pp. 3040-3048.
Joslin
Camporez, J. P., et al. “Mechanism By Which Arylamine -Acetyltransferase 1 Ablation Causes Insulin Resistance In Mice.”. Proceedings Of The National Academy Of Sciences Of The United States Of America, pp. E11285-E11292.
Yale
Rohatgi, K. W., et al. “Relationships Between Consumption Of Ultra-Processed Foods, Gestational Weight Gain And Neonatal Outcomes In A Sample Of Us Pregnant Women.”. Peerj, p. e4091.
WUSTL
Sweeney, P., et al. “Appetite Suppressive Role Of Medial Septal Glutamatergic Neurons.”. Proceedings Of The National Academy Of Sciences Of The United States Of America, pp. 13816-13821.
Einstein
Kim, S. P., et al. “Sclerostin Influences Body Composition By Regulating Catabolic And Anabolic Metabolism In Adipocytes.”. Proceedings Of The National Academy Of Sciences Of The United States Of America, pp. E11238-E11247.
JHU-UMD
Fried, M., et al. “A Novel Approach To Glycemic Control In Type 2 Diabetes Mellitus, Partial Jejunal Diversion: Pre-Clinical To Clinical Pathway.”. Bmj Open Diabetes Research & Care, p. e000431.
Michigan
Rubinow, K. B. “Estrogens And Body Weight Regulation In Men.”. Advances In Experimental Medicine And Biology, pp. 285-313.
Washington
Hevener, A. L., et al. “The Role Of Skeletal Muscle Estrogen Receptors In Metabolic Homeostasis And Insulin Sensitivity.”. Advances In Experimental Medicine And Biology, pp. 257-284.
UCSD-UCLA
 Multicenter
Bogdani, M., et al. “Structural Abnormalities In Islets From Very Young Children With Cystic Fibrosis May Contribute To Cystic Fibrosis-Related Diabetes.”. Scientific Reports, p. 17231.
Vanderbilt
Washington
Méndez-Giráldez, R., et al. “Gwas Of The Electrocardiographic Qt Interval In Hispanics/Latinos Generalizes Previously Identified Loci And Identifies Population-Specific Signals.”. Scientific Reports, p. 17075.
UCSD-UCLA
Garcia-Galiano, D., et al. “Pi3Kα Inactivation In Leptin Receptor Cells Increases Leptin Sensitivity But Disrupts Growth And Reproduction.”. Jci Insight.
Michigan
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