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Stratigopoulos, G., et al. “Hypomorphism Of Fto And Rpgrip1L Causes Obesity In Mice.”. The Journal Of Clinical Investigation, pp. 1897-910.
Columbia
Lee, B. -C., et al. “Adipose Natural Killer Cells Regulate Adipose Tissue Macrophages To Promote Insulin Resistance In Obesity.”. Cell Metabolism, pp. 685-98.
Joslin
Lagerlöf, O., et al. “The Nutrient Sensor Ogt In Pvn Neurons Regulates Feeding.”. Science (New York, N.y.), pp. 1293-6.
JHU-UMD
Jang, C., et al. “A Branched-Chain Amino Acid Metabolite Drives Vascular Fatty Acid Transport And Causes Insulin Resistance.”. Nature Medicine, pp. 421-6.
Penn
Boston Area
Arnes, L., et al. “Βlinc1 Encodes A Long Noncoding Rna That Regulates Islet Β-Cell Formation And Function.”. Genes & Development, pp. 502-7.
Columbia
Magkos, F., et al. “Effects Of Moderate And Subsequent Progressive Weight Loss On Metabolic Function And Adipose Tissue Biology In Humans With Obesity.”. Cell Metabolism, pp. 591-601.
WUSTL
Lee, J., et al. “Loss Of Adipose Fatty Acid Oxidation Does Not Potentiate Obesity At Thermoneutrality.”. Cell Reports, pp. 1308-1316.
JHU-UMD
Ramakrishnan, S. K., et al. “Hif2Α Is An Essential Molecular Brake For Postprandial Hepatic Glucagon Response Independent Of Insulin Signaling.”. Cell Metabolism, pp. 505-16.
Michigan
Khamaisi, M., et al. “Pkcδ Inhibition Normalizes The Wound-Healing Capacity Of Diabetic Human Fibroblasts.”. The Journal Of Clinical Investigation, pp. 837-53.
Joslin
Schechter, C. B., et al. “Costs And Effects Of A Telephonic Diabetes Self-Management Support Intervention Using Health Educators.”. Journal Of Diabetes And Its Complications, pp. 300-5.
Einstein
Rajagopal, R., et al. “Functional Deficits Precede Structural Lesions In Mice With High-Fat Diet-Induced Diabetic Retinopathy.”. Diabetes, pp. 1072-84.
WUSTL