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Center Publications

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Todd, W. D., et al. “A Hypothalamic Circuit For The Circadian Control Of Aggression.”. Nature Neuroscience, pp. 717-724.
Boston Area
Tikkanen, E., et al. “Associations Of Fitness, Physical Activity, Strength, And Genetic Risk With Cardiovascular Disease: Longitudinal Analyses In The Uk Biobank Study.”. Circulation, pp. 2583-2591.
Stanford
Muse, E. D., et al. “Cell-Specific Discrimination Of Desmosterol And Desmosterol Mimetics Confers Selective Regulation Of Lxr And Srebp In Macrophages.”. Proceedings Of The National Academy Of Sciences Of The United States Of America, pp. E4680-E4689.
UCSD-UCLA
Shang, W., et al. “Genome-Wide Crispr Screen Identifies Fam49B As A Key Regulator Of Actin Dynamics And T Cell Activation.”. Proceedings Of The National Academy Of Sciences Of The United States Of America, pp. E4051-E4060.
UCSF
Schloss, J., et al. “Hla-B*39:06 Efficiently Mediates Type 1 Diabetes In A Mouse Model Incorporating Reduced Thymic Insulin Expression.”. Journal Of Immunology (Baltimore, Md. : 1950), pp. 3353-3363.
Einstein
Doonan, L. M., et al. “Can Modulators Of Apolipoproteinb Biogenesis Serve As An Alternate Target For Cholesterol-Lowering Drugs?”. Biochimica Et Biophysica Acta. Molecular And Cell Biology Of Lipids, pp. 762-771.
Einstein
Mottalib, A., et al. “Effects Of Nutrition Therapy On Hba1C And Cardiovascular Disease Risk Factors In Overweight And Obese Patients With Type 2 Diabetes.”. Nutrition Journal, p. 42.
Joslin
Desai, P. B., et al. “Ift25 Is Not A Cystic Kidney Disease Gene But Is Required For Early Steps Of Kidney Development.”. Mechanisms Of Development, pp. 10-17.
UMass
Capozzi, M. E., et al. “Palmitic Acid Induces Müller Cell Inflammation That Is Potentiated By Co-Treatment With Glucose.”. Scientific Reports, p. 5459.
Vanderbilt
Štěpánková, M., et al. “Methylindoles And Methoxyindoles Are Agonists And Antagonists Of Human Aryl Hydrocarbon Receptor.”. Molecular Pharmacology, pp. 631-644.
Einstein
Aguayo-Mazzucato, C., et al. “T Induces Both Markers Of Maturation And Aging In Pancreatic Β-Cells.”. Diabetes, pp. 1322-1331.
Joslin
Kycia, I., et al. “A Common Type 2 Diabetes Risk Variant Potentiates Activity Of An Evolutionarily Conserved Islet Stretch Enhancer And Increases C2Cd4A And C2Cd4B Expression.”. American Journal Of Human Genetics, pp. 620-635.
Michigan