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Can modulators of apolipoproteinB biogenesis serve as an alternate target for cholesterol-lowering drugs?

Citation
Doonan, L. M., et al. “Can Modulators Of Apolipoproteinb Biogenesis Serve As An Alternate Target For Cholesterol-Lowering Drugs?”. Biochimica Et Biophysica Acta. Molecular And Cell Biology Of Lipids, pp. 762-771.
Center Albert Einstein College of Medicine
Author Lynley M Doonan, Edward A Fisher, Jeffrey L Brodsky
Keywords ApolipoproteinB, Autophagy, Cardiovascular disease, cholesterol, Endoplasmic Reticulum Associated Degradation (ERAD), Very Low Density Lipoprotein (VLDL)
Abstract

Understanding the molecular defects underlying cardiovascular disease is necessary for the development of therapeutics. The most common method to lower circulating lipids, which reduces the incidence of cardiovascular disease, is statins, but other drugs are now entering the clinic, some of which have been approved. Nevertheless, patients cannot tolerate some of these therapeutics, the drugs are costly, and/or the treatments are approved for only rare forms of disease. Efforts to find alternative treatments have focused on other factors, such as apolipoproteinB (apoB), which transports cholesterol in the blood stream. The levels of apoB are regulated by endoplasmic reticulum (ER) associated degradation as well as by a post ER degradation pathway in model systems, and we suggest that these events provide novel therapeutic targets. We discuss first how cardiovascular disease arises and how cholesterol is regulated, and then summarize the mechanisms of action of existing treatments for cardiovascular disease. We then review the apoB biosynthetic pathway, focusing on steps that might be amenable to therapeutic interventions.

Year of Publication
2018
Journal
Biochimica et biophysica acta. Molecular and cell biology of lipids
Volume
1863
Issue
7
Number of Pages
762-771
Date Published
12/2018
ISSN Number
1388-1981
DOI
10.1016/j.bbalip.2018.03.010
Alternate Journal
Biochim Biophys Acta Mol Cell Biol Lipids
PMID
29627384
PMCID
PMC5953829
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