Multicenter Publications

Multicenter	Temple, K. A., et al. “Association Of Habitual Daily Physical Activity With Glucose Tolerance And Β-Cell Function In Adults With Impaired Glucose Tolerance Or Recently Diagnosed Type 2 Diabetes From The Restoring Insulin Secretion (Rise) Study.”. Diabetes Care, pp. 1521-1529.	DOI PubMed PMC	Washington
			Multicenter	Temple, K. A., et al. “Association Of Habitual Daily Physical Activity With Glucose Tolerance And Β-Cell Function In Adults With Impaired Glucose Tolerance Or Recently Diagnosed Type 2 Diabetes From The Restoring Insulin Secretion (Rise) Study.”. Diabetes Care, pp. 1521-1529.	DOI PubMed PMC	Yale
Multicenter	Aguayo-Mazzucato, C., et al. “Acceleration Of Β Cell Aging Determines Diabetes And Senolysis Improves Disease Outcomes.”. Cell Metabolism, pp. 129-142.e4.	DOI PubMed PMC	Boston Area
			Multicenter	Aguayo-Mazzucato, C., et al. “Acceleration Of Β Cell Aging Determines Diabetes And Senolysis Improves Disease Outcomes.”. Cell Metabolism, pp. 129-142.e4.	DOI PubMed PMC	Joslin
Multicenter	Flannick, J., et al. “Exome Sequencing Of 20,791 Cases Of Type 2 Diabetes And 24,440 Controls.”. Nature, pp. 71-76.	DOI PubMed PMC	Boston Area
			Multicenter	Flannick, J., et al. “Exome Sequencing Of 20,791 Cases Of Type 2 Diabetes And 24,440 Controls.”. Nature, pp. 71-76.	DOI PubMed PMC	UCSD-UCLA
			Multicenter	Flannick, J., et al. “Exome Sequencing Of 20,791 Cases Of Type 2 Diabetes And 24,440 Controls.”. Nature, pp. 71-76.	DOI PubMed PMC	Chicago
Multicenter	Ji, Y., et al. “Toll-Like Receptors Tlr2 And Tlr4 Block The Replication Of Pancreatic Β Cells In Diet-Induced Obesity.”. Nature Immunology, pp. 677-686.	DOI PubMed PMC	Michigan
			Multicenter	Ji, Y., et al. “Toll-Like Receptors Tlr2 And Tlr4 Block The Replication Of Pancreatic Β Cells In Diet-Induced Obesity.”. Nature Immunology, pp. 677-686.	DOI PubMed PMC	Joslin
Featured Multicenter	Gregory, J. M., et al. “Iatrogenic Hyperinsulinemia, Not Hyperglycemia, Drives Insulin Resistance In Type 1 Diabetes As Revealed By Comparison With Gck-Mody (Mody2).”. Diabetes, pp. 1565-1576.	DOI PubMed PMC	Vanderbilt
			Featured Multicenter	Gregory, J. M., et al. “Iatrogenic Hyperinsulinemia, Not Hyperglycemia, Drives Insulin Resistance In Type 1 Diabetes As Revealed By Comparison With Gck-Mody (Mody2).”. Diabetes, pp. 1565-1576.	DOI PubMed PMC	Chicago
Multicenter	Guan, M., et al. “Genome-Wide Association Study Identifies Novel Loci For Type 2 Diabetes-Attributed End-Stage Kidney Disease In African Americans.”. Human Genomics, p. 21.	DOI PubMed PMC	UCSD-UCLA